lentivirus encoding mouse trim56 (Genechem)
86
Structured Review
Genechem
lentivirus encoding mouse trim56
Lentivirus Encoding Mouse Trim56, supplied by Genechem, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/lentivirus+encoding+mouse+trim56/hairpin+rna+short/pm42155789-55-10-23
Average 86 stars, based on 1 article reviews
Lentivirus Encoding Mouse Trim56, supplied by Genechem, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/lentivirus+encoding+mouse+trim56/hairpin+rna+short/pm42155789-55-10-23
Average 86 stars, based on 1 article reviews
lentivirus encoding mouse trim56 - by Bioz Stars,
2026-09
86/100 stars
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Related Articles
Over Expression:Article Title: TRIM56 inhibits M1 macrophage polarization and mitigates sepsis-induced acute lung injury via promoting STING ubiquitination-degradation. Article Snippet: Sepsis-induced acute lung injury (ALI) is a life-threatening clinical disorder with high mortality, and its pathogenesis is closely linked to excessive M1 macrophage polarization and overactivation of the STING signaling pathway.. TRIM56, an E3 ubiquitin ligase, is known to participate in immune and inflammatory regulation; however, its specific role and underlying molecular mechanism in sepsis-induced ALI remain largely unclear.. Here, we aimed to determine whether TRIM56 alleviates sepsis-induced ALI by inhibiting M1 macrophage polarization via promoting STING ubiquitination and proteasomal degradation. Injection:Article Title: TRIM56 inhibits M1 macrophage polarization and mitigates sepsis-induced acute lung injury via promoting STING ubiquitination-degradation. Article Snippet: Sepsis-induced acute lung injury (ALI) is a life-threatening clinical disorder with high mortality, and its pathogenesis is closely linked to excessive M1 macrophage polarization and overactivation of the STING signaling pathway.. TRIM56, an E3 ubiquitin ligase, is known to participate in immune and inflammatory regulation; however, its specific role and underlying molecular mechanism in sepsis-induced ALI remain largely unclear.. Here, we aimed to determine whether TRIM56 alleviates sepsis-induced ALI by inhibiting M1 macrophage polarization via promoting STING ubiquitination and proteasomal degradation. |